Fig. 2

Silencing of TRPM2-AS inhibits OC cell proliferation and promotes cell apoptosis. A PCR analysis was employed to evaluate knockdown efficacy of TRPM2-AS in OC cells. B–E In response to TRPM2-AS depletion, OC cell viability (B), proliferation (C), and apoptosis (D, E) were measured using CCK-8 assays (B), colony forming assays (C), TUNEL assays (D), and flow cytometry analyses (E). F Protein expression of apoptotic markers was quantified utilizing western blotting with normalization to GAPDH. **p < 0.01